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HOME > News & Topics > Research > FY 2019 > Complement and inflammasome overactivation mediates paroxysmal nocturnal hemoglobinuria with autoinflammation.

Complement and inflammasome overactivation mediates paroxysmal nocturnal hemoglobinuria with autoinflammation.

Yoshiko Murakami, Taroh Kinoshita (Immunoglycobiology, IFReC & RIMD) and their research group report the mechanistic basis of atypical paroxysmal nocturnal hemoglobinuria (PNH) caused by germline mutation plus somatic loss of PIGT gene on chromosome 20q.

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External Article


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Immunoglycobiology Lab

WPI-Immunology Frontier Research Center

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